作业帮求助大家帮我翻译一段英语
来源：学生作业帮 编辑：作业帮 分类：英语作业 时间：2024/07/05 23:37:14
求助大家帮我翻译一段英语
REGULARITIES OF PARANEOPLASTIC DISORDERS IN EDEMATOUS INFILTRATIVE
BREAST CANCER
Data on complex clinical and laboratory studies of immune status, hemostasis as well as metabolic shifts in patients with edematous infiltrative breast cancer are presented. Development
of β-dependent and T-dependent immunodeficiency conditions and decrease in NK-blood cells are considered to be the leading factors in development of malignant cells elimination disorder and transition of transformation stage to promotion one. Decrease in coagulation blood potential, activation of fibrinolisis system which are typical for patients with edematous infiltrative
breast cancer indicate the development of “hypocoagulation consumption”. They are also proved to be prognostically unfavorable signs due to the possibility of albuminolysis intensification because of urokinase and tissue plasminogen activators influence expressed by neoplasia cells, structural disorganization of tissue in areas distant from the neoplasia focus as well as formation of “site” for metastatic cells. Intensification of production of cytokines: IL-1, TNFα,GCSF is typical for edematous infiltrative breast cancer and it is most probably of compensatory
and adaptation nature.
不要用机器翻译
REGULARITIES OF PARANEOPLASTIC DISORDERS IN EDEMATOUS INFILTRATIVE
BREAST CANCER
Data on complex clinical and laboratory studies of immune status, hemostasis as well as metabolic shifts in patients with edematous infiltrative breast cancer are presented. Development
of β-dependent and T-dependent immunodeficiency conditions and decrease in NK-blood cells are considered to be the leading factors in development of malignant cells elimination disorder and transition of transformation stage to promotion one. Decrease in coagulation blood potential, activation of fibrinolisis system which are typical for patients with edematous infiltrative
breast cancer indicate the development of “hypocoagulation consumption”. They are also proved to be prognostically unfavorable signs due to the possibility of albuminolysis intensification because of urokinase and tissue plasminogen activators influence expressed by neoplasia cells, structural disorganization of tissue in areas distant from the neoplasia focus as well as formation of “site” for metastatic cells. Intensification of production of cytokines: IL-1, TNFα,GCSF is typical for edematous infiltrative breast cancer and it is most probably of compensatory
and adaptation nature.
不要用机器翻译
渗透性水肿乳腺癌的类肿瘤失调的规律
在免疫状况的临床以及实验室研究数据,渗透性水肿乳腺癌的患者的止血以及新陈代谢转换是先露的,β淋巴细胞和T淋巴细胞在NK血细胞的免疫缺陷状况和减少被认为是恶性细胞消除混乱发展的主要因素以及转换过程中的过渡,促进其中一方.渗透性水肿乳腺癌患者典型的血液凝结潜力下降,纤维蛋白溶解活跃揭示了“低凝结消耗”的发展.在病情预断中,它们由于蛋白分解强化的可能性而被证明是不乐观的,因为蛋白分解强化是由瘤形成细胞表达的尿激酶以及组织血纤维蛋白溶酶原活化质影响,组织结构破坏的地方离瘤形成中心和转移性细胞的形成地距离遥远引起的.
细胞因子生产加强,细胞因子：IL-1,TNFα,GCSF是渗透性水肿乳腺癌的典型细胞因子,它最有可能补偿以及改造其本质.
好累.我是绝对鄙视机翻的,楼主可以放心.不过不够专业.毕竟我不学这块啦.楼主要是没有其他更好的答案就采纳我的吧~=_=..
在免疫状况的临床以及实验室研究数据,渗透性水肿乳腺癌的患者的止血以及新陈代谢转换是先露的,β淋巴细胞和T淋巴细胞在NK血细胞的免疫缺陷状况和减少被认为是恶性细胞消除混乱发展的主要因素以及转换过程中的过渡,促进其中一方.渗透性水肿乳腺癌患者典型的血液凝结潜力下降,纤维蛋白溶解活跃揭示了“低凝结消耗”的发展.在病情预断中,它们由于蛋白分解强化的可能性而被证明是不乐观的,因为蛋白分解强化是由瘤形成细胞表达的尿激酶以及组织血纤维蛋白溶酶原活化质影响,组织结构破坏的地方离瘤形成中心和转移性细胞的形成地距离遥远引起的.
细胞因子生产加强,细胞因子：IL-1,TNFα,GCSF是渗透性水肿乳腺癌的典型细胞因子,它最有可能补偿以及改造其本质.
好累.我是绝对鄙视机翻的,楼主可以放心.不过不够专业.毕竟我不学这块啦.楼主要是没有其他更好的答案就采纳我的吧~=_=..